Mechanisms of Hyperbaric Oxygen Induced Brain Protection,
Role: Consultant,
National Institute of Neurological Disorders and Stroke/NIH/DHHS,
(06/2010 - 05/2016)
Status: Closed
LTQ-Orbitrap-XL-ETD Mass Spectrometer for LLU Mass Spectrometry Facility,
Role: Major Users,
National Center for Research Resources/NIH/DHHS,
(04/2010 - 04/2011)
Status: Closed
Mechanisms of G-CSF-Induced Neuroprotection,
Role: Significant Contributor,
National Institute of Neurological Disorders and Stroke/NIH/DHHS,
(08/2009 - 07/2015)
Status: Closed
Pain and Hypoxia in Premature Neonates,
Role: Consultant,
National Institute of Nursing Research/NIH/DHHS,
(07/2009 - 04/2014)
Status: Closed
Mechanisms for adaptation to oxidative stress in Porphyromonas gingivalis,
Role: Consultant,
National Institute of Dental and Craniofacial Research/NIH/DHHS,
(06/2009 - 03/2015)
Status: Closed
NIH-LLU Initiative for Maximizing Student Diversity,
Role: Mentor,
National Institute of General Medical Sciences/NIH/DHHS,
(04/2009 - 03/2013)
Status: Closed
Base pairing and mutagenesis,
Role: Principal Investigator,
National Institutes of Health/DHHS,
(12/2006 - 11/2009)
Status: Closed
Chemical pathology of 5-aza-2'-deoxycytidine (Supplemental for Jason Herring),
Role: Principal Investigator,
National Institutes of Health/DHHS,
(05/2006 - 04/2009)
Status: Closed
Hydrolytic and Free Radical Mediated DNA Damage,
Role: Principal Investigator,
National Institutes of Health/DHHS,
(04/2006 - 03/2007)
Status: Closed
Prenatal cocaine exposure and cardiac programming,
Role: Consultant,
National Heart, Lung, and Blood Institute/NIH/DHHS,
(01/2006 - 12/2010)
Status: Closed
Base pairing and mutagenesis,
Role: Principal Investigator,
National Institutes of Health/DHHS,
(12/2005 - 11/2006)
Status: Closed
Chemical pathology of 5-aza-2'-deoxycytidine (Supplement for Jason Herring),
National Institutes of Health/DHHS,
(07/2005 - 06/2006)
Status: Closed
Infidelity of cytosine methylation and human cancer,
National Cancer Institute/NIH/DHHS,
(05/2005 - 04/2011)
Status: Closed
Hydrolytic and free radical mediated DNA damage (GM050351-10),
National Institutes of Health/DHHS,
(04/2005 - 03/2006)
Status: Closed
Damaged DNA recognition as a cancer avoidance mechanism,
National Institutes of Health/DHHS,
(02/2005 - 07/2010)
Status: Closed
Base pairing and mutagenesis,
National Institutes of Health/DHHS,
(12/2004 - 11/2005)
Status: Closed
Hydrolytic and free radical mediated DNA damage (GM050351,
National Institute of General Medical Sciences/NIH/DHHS,
(04/2004 - 03/2005)
Status: Closed
Base pairing and mutagenesis,
National Institute of General Medical Sciences/NIH/DHHS,
(12/2003 - 11/2009)
Status: Closed
Chemical pathology of 5-aza-2'-deoxycytidine,
National Institutes of Health/DHHS,
(07/2003 - 04/2010)
Status: Closed
Chemical pathology of 5-aza-2'-deoxycytidine,
Role: PD/PI,
National Institutes of Health/DHHS,
(07/2003 - 04/2010)
Status: Closed
Infidelity of Cytosine Methylation and Human Cancer,
National Cancer Institute/NIH/DHHS,
(05/2003 - 04/2004)
Status: Closed
Carcinogenic pyrimidines at a model replication fork,
National Institutes of Health/DHHS,
(06/2002 - 05/2004)
Status: Closed
Infidelity of cytosine methylation and human cancer,
Role: Key,
National Institutes of Health/DHHS,
(05/2002 - 04/2003)
Status: Closed
Infidelity of cytosine methylation and human cancer,
National Cancer Institute/NIH/DHHS,
(08/2001 - 04/2004)
Status: Closed
Base pairing mutagenesis,
National Institutes of Health/DHHS,
(08/2001 - 07/2002)
Status: Closed
Hydrolytic and free radical mediated DNA damage,
National Institutes of Health/DHHS,
(04/2001 - 03/2002)
Status: Closed
Hydrolytic and free radical mediated DNA damage,
National Institutes of Health/DHHS,
(01/1994 - 03/2009)
Status: Closed
Hydrolytic and free radical mediated DNA damage,
National Institutes of Health/DHHS,
(01/1994 - 03/2003)
Status: Closed
Hydrolytic and Free Radical Mediated DNA Damage,
Role: PD/PI,
National Institutes of Health/DHHS,
(01/1994 - 12/2012)
Status: Closed
Base pairing and mutagenesis,
National Institutes of Health/DHHS,
(12/1988 - 07/2003)
Status: Closed
Carcinogen pyramidines at a model replication fork,
National Institutes of Health/DHHS,
Status: Closed
Decitabine: Mechanism of toxicity, mutagenicity and demethylation,
National Institutes of Health/DHHS,
Status: Closed
Hydrolytic and Free Radical Mediated DNA Damage,
Role: Principal Investigator,
National Institutes of Health/DHHS,
Status: Closed